Luque Contreras, Diana <br/><br/>
Hindlimb claudication reflects impaired nitric oxide-dependent revascularization after ischemia.  [electronic resource] 

 -  Vascular pharmacology  Jan 2007 
 - 10-5 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1537-1891 
<br/><br/><label>Standard No.: </label>10.1016/j.vph.2006.06.017  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Arterial Occlusive Diseases--complications<br/>Biomechanical Phenomena<br/>Blood Flow Velocity--drug effects<br/>Disease Models, Animal<br/>Enzyme Induction<br/>Enzyme Inhibitors--pharmacology<br/>Gait--drug effects<br/>Hindlimb<br/>Intermittent Claudication--etiology<br/>Ischemia--etiology<br/>Locomotion--drug effects<br/>Male<br/>Muscle, Skeletal--blood supply<br/>NG-Nitroarginine Methyl Ester--pharmacology<br/>Neovascularization, Physiologic<br/>Nitric Oxide--metabolism<br/>Nitric Oxide Synthase Type III--antagonists & inhibitors<br/>Peripheral Vascular Diseases--complications<br/>RNA, Messenger--biosynthesis<br/>Rats<br/>Rats, Wistar<br/>Recovery of Function<br/>Time Factors