Lu, Ting <br/><br/>
Estrogen receptor alpha regulates matrix metalloproteinase-13 promoter activity primarily through the AP-1 transcriptional regulatory site.  [electronic resource] 

 -  Biochimica et biophysica acta  Aug 2006 
 - 719-31 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0006-3002 
<br/><br/><label>Standard No.: </label>10.1016/j.bbadis.2006.06.007  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Blotting, Western<br/>Cells, Cultured<br/>Collagenases--genetics<br/>DNA--metabolism<br/>Estradiol--pharmacology<br/>Estrogen Receptor alpha--agonists<br/>Female<br/>Gene Expression Regulation--drug effects<br/>Immunohistochemistry<br/>Matrix Metalloproteinase 13<br/>Promoter Regions, Genetic--drug effects<br/>RNA, Messenger--genetics<br/>Rabbits<br/>Selective Estrogen Receptor Modulators--pharmacology<br/>Sequence Deletion<br/>Transcription Factor AP-1--genetics<br/>Transfection<br/>Uterus--cytology