Azevedo, Luísa <br/><br/>
Epistatic interactions: how strong in disease and evolution?  [electronic resource] 

 -  Trends in genetics : TIG  Nov 2006 
 - 581-5 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Review 
<br/><br/><label>ISSN: </label>0168-9525 
<br/><br/><label>Standard No.: </label>10.1016/j.tig.2006.08.001  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adaptation, Biological<br/>Adaptor Proteins, Signal Transducing<br/>Alleles<br/>Animals<br/>Biological Evolution<br/>Carrier Proteins--genetics<br/>Cytoskeletal Proteins--genetics<br/>Dosage Compensation, Genetic<br/>Epistasis, Genetic<br/>Eye Proteins--genetics<br/>Genetic Diseases, Inborn--genetics<br/>Glycoproteins--genetics<br/>Group II Chaperonins<br/>Humans<br/>Models, Molecular<br/>Molecular Chaperones--genetics<br/>MutL Protein Homolog 1<br/>Mutation<br/>Nuclear Proteins--genetics<br/>Pan troglodytes--genetics<br/>Recombination, Genetic<br/>Transcription Factors--genetics<br/>Trypsin<br/>Trypsinogen--genetics<br/>AIRE Protein