Cardioprotection via activation of protein kinase C-delta depends on modulation of the reverse mode of the Na+/Ca2+ exchanger. [electronic resource]
- Circulation Jul 2006
- I226-32 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1524-4539
10.1161/CIRCULATIONAHA.105.000570 doi
Anesthetics, Inhalation--pharmacology Aniline Compounds--pharmacology Animals Calcium Signaling--drug effects Cardiotonic Agents--pharmacology Enzyme Activation Glucose--pharmacology Heart Ventricles--drug effects In Vitro Techniques Ischemic Preconditioning, Myocardial--methods Male Methyl Ethers--pharmacology Myocardial Ischemia--enzymology Myocardial Reperfusion Injury--enzymology Phenyl Ethers--pharmacology Phosphorylation--drug effects Phosphoserine--analysis Protein Kinase C-delta--physiology Protein Processing, Post-Translational--drug effects Protein Transport--drug effects Rats Rats, Wistar Sarcolemma--enzymology Sevoflurane Signal Transduction--drug effects Sodium-Calcium Exchanger--antagonists & inhibitors Thiourea--analogs & derivatives