Sipilä, Sampsa T <br/><br/>
Intrinsic bursting of immature CA3 pyramidal neurons and consequent giant depolarizing potentials are driven by a persistent Na+ current and terminated by a slow Ca2+ -activated K+ current.  [electronic resource] 

 -  The European journal of neuroscience  May 2006 
 - 2330-8 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0953-816X 
<br/><br/><label>Standard No.: </label>10.1111/j.1460-9568.2006.04757.x  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Animals, Newborn<br/>Dose-Response Relationship, Drug<br/>Dose-Response Relationship, Radiation<br/>Electric Stimulation--methods<br/>Excitatory Amino Acid Antagonists--pharmacology<br/>Hippocampus--cytology<br/>In Vitro Techniques<br/>Membrane Potentials--drug effects<br/>Metals, Heavy--pharmacology<br/>Neurons--physiology<br/>Patch-Clamp Techniques--methods<br/>Phenytoin--pharmacology<br/>Potassium--pharmacology<br/>Potassium Channels, Calcium-Activated--physiology<br/>Rats<br/>Rats, Wistar<br/>Sodium Channel Blockers--pharmacology<br/>Sodium Channels--physiology<br/>Tetrodotoxin--pharmacology