Leinhase, Iris <br/><br/>
Pharmacological complement inhibition at the C3 convertase level promotes neuronal survival, neuroprotective intracerebral gene expression, and neurological outcome after traumatic brain injury.  [electronic resource] 

 -  Experimental neurology  Jun 2006 
 - 454-64 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0014-4886 
<br/><br/><label>Standard No.: </label>10.1016/j.expneurol.2006.01.033  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Behavior, Animal--drug effects<br/>Blotting, Western--methods<br/>Brain--drug effects<br/>Brain Injuries--drug therapy<br/>Cell Survival--drug effects<br/>Complement System Proteins--genetics<br/>Disease Models, Animal<br/>Gene Expression--drug effects<br/>Immunohistochemistry--methods<br/>Male<br/>Mice<br/>Mice, Inbred C57BL<br/>Neurons--drug effects<br/>Neuroprotective Agents--administration & dosage<br/>Proto-Oncogene Proteins c-bcl-2--genetics<br/>Psychomotor Performance--drug effects<br/>RNA, Messenger--metabolism<br/>Receptors, Complement--administration & dosage<br/>Receptors, Complement 3b<br/>Reverse Transcriptase Polymerase Chain Reaction--methods<br/>Time Factors<br/>Trauma Severity Indices