Black, Joel A <br/><br/>
Remyelination of dorsal column axons by endogenous Schwann cells restores the normal pattern of Nav1.6 and Kv1.2 at nodes of Ranvier.  [electronic resource] 

 -  Brain : a journal of neurology  May 2006 
 - 1319-29 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S. 
<br/><br/><label>ISSN: </label>1460-2156 
<br/><br/><label>Standard No.: </label>10.1093/brain/awl057  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Axons--physiology<br/>Demyelinating Diseases--chemically induced<br/>Ethidium<br/>Kv1.2 Potassium Channel--metabolism<br/>Myelin Sheath--physiology<br/>NAV1.6 Voltage-Gated Sodium Channel<br/>Nerve Regeneration--physiology<br/>Nerve Tissue Proteins--metabolism<br/>Ranvier's Nodes--metabolism<br/>Rats<br/>Rats, Sprague-Dawley<br/>Schwann Cells--transplantation<br/>Sodium Channels--metabolism<br/>Spinal Cord--physiology<br/>Spinal Cord Diseases--chemically induced