Satoh, Kimio <br/><br/>
Important role of endogenous erythropoietin system in recruitment of endothelial progenitor cells in hypoxia-induced pulmonary hypertension in mice.  [electronic resource] 

 -  Circulation  Mar 2006 
 - 1442-50 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1524-4539 
<br/><br/><label>Standard No.: </label>10.1161/CIRCULATIONAHA.105.583732  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Bone Marrow Transplantation<br/>Cell Movement<br/>Cells, Cultured--cytology<br/>Chronic Disease<br/>Endothelial Cells--pathology<br/>Endothelium--pathology<br/>Endothelium, Vascular--pathology<br/>Enzyme Activation<br/>Erythroid Precursor Cells--metabolism<br/>Erythropoietin--physiology<br/>GATA1 Transcription Factor--physiology<br/>Heart Failure--etiology<br/>Hematopoietic Stem Cells--physiology<br/>Hypertension, Pulmonary--etiology<br/>Hypertrophy, Right Ventricular--etiology<br/>Hypoxia--complications<br/>Lung--blood supply<br/>Male<br/>Mice<br/>Mice, Knockout<br/>Mice, Transgenic<br/>Muscle, Smooth, Vascular--pathology<br/>Nitric Oxide Synthase Type III--metabolism<br/>Organ Specificity<br/>Radiation Chimera<br/>Receptor, TIE-2--genetics<br/>Receptors, Erythropoietin--deficiency<br/>Systole<br/>Ventricular Dysfunction, Right--etiology