Baker, David J <br/><br/>
Nitric oxide synthase inhibition reduces O2 cost of force development and spares high-energy phosphates following contractions in pump-perfused rat hindlimb muscles.  [electronic resource] 

 -  Experimental physiology  May 2006 
 - 581-9 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0958-0670 
<br/><br/><label>Standard No.: </label>10.1113/expphysiol.2005.032698  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adenosine--administration & dosage<br/>Adenosine Triphosphate--metabolism<br/>Animals<br/>Drug Combinations<br/>Hindlimb--physiology<br/>Infusion Pumps<br/>Male<br/>Muscle Contraction--physiology<br/>Muscle, Skeletal--blood supply<br/>NG-Nitroarginine Methyl Ester--administration & dosage<br/>Nitric Oxide Synthase--antagonists & inhibitors<br/>Oxygen--metabolism<br/>Oxygen Consumption--physiology<br/>Perfusion--methods<br/>Rats<br/>Rats, Sprague-Dawley<br/>Stress, Mechanical<br/>Vasodilator Agents--administration & dosage