Scruggs, Sarah B <br/><br/>
Partial replacement of cardiac troponin I with a non-phosphorylatable mutant at serines 43/45 attenuates the contractile dysfunction associated with PKCepsilon phosphorylation.  [electronic resource] 

 -  Journal of molecular and cellular cardiology  Apr 2006 
 - 465-73 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0022-2828 
<br/><br/><label>Standard No.: </label>10.1016/j.yjmcc.2005.12.009  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Aging--genetics<br/>Amino Acid Substitution<br/>Animals<br/>Cell Line<br/>Heart Failure--genetics<br/>Mice<br/>Mice, Transgenic<br/>Myocardial Contraction--genetics<br/>Myocardium--metabolism<br/>Phosphorylation<br/>Protein Kinase C-epsilon--metabolism<br/>Protein Processing, Post-Translational--genetics<br/>Troponin I--genetics<br/>Ventricular Function, Left--genetics