Amelio, Antonio L <br/><br/>
Deacetylation of the herpes simplex virus type 1 latency-associated transcript (LAT) enhancer and a decrease in LAT abundance precede an increase in ICP0 transcriptional permissiveness at early times postexplant.  [electronic resource] 

 -  Journal of virology  Feb 2006 
 - 2063-8 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0022-538X 
<br/><br/><label>Standard No.: </label>10.1128/JVI.80.4.2063-2068.2006  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Acetylation<br/>Animals<br/>Enhancer Elements, Genetic<br/>Female<br/>Ganglia, Spinal--virology<br/>Gene Expression Regulation, Viral<br/>Herpesvirus 1, Human--genetics<br/>Histones--metabolism<br/>Immediate-Early Proteins--genetics<br/>Mice<br/>Promoter Regions, Genetic<br/>RNA, Untranslated--analysis<br/>RNA, Viral--analysis<br/>Transcription, Genetic<br/>Ubiquitin-Protein Ligases--genetics<br/>Virus Latency--physiology