de Voer, G <br/><br/>
Deletion of the Caenorhabditis elegans homologues of the CLN3 gene, involved in human juvenile neuronal ceroid lipofuscinosis, causes a mild progeric phenotype.  [electronic resource] 

 -  Journal of inherited metabolic disease  2005 
 - 1065-80 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0141-8955 
<br/><br/><label>Standard No.: </label>10.1007/s10545-005-0125-7  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Alleles<br/>Animals<br/>Caenorhabditis elegans<br/>Carbohydrate Metabolism<br/>Cosmids<br/>DNA Primers<br/>Disease Models, Animal<br/>Female<br/>Gene Deletion<br/>Genes, Reporter<br/>Green Fluorescent Proteins--metabolism<br/>Lysosomes--metabolism<br/>Male<br/>Membrane Glycoproteins--metabolism<br/>Microscopy, Electron<br/>Microscopy, Fluorescence<br/>Models, Genetic<br/>Models, Statistical<br/>Molecular Chaperones--metabolism<br/>Mutation<br/>Mutation, Missense<br/>Neurodegenerative Diseases<br/>Neuronal Ceroid-Lipofuscinoses--diagnosis<br/>Neurons--metabolism<br/>Phenotype<br/>Progeria--genetics<br/>Promoter Regions, Genetic<br/>Recombinant Fusion Proteins--metabolism<br/>Time Factors<br/>Transgenes