Lee, Shin-Da <br/><br/>
Porphyromonas gingivalis-related cardiac cell apoptosis was majorly co-activated by p38 and extracellular signal-regulated kinase pathways.  [electronic resource] 

 -  Journal of periodontal research  Feb 2006 
 - 39-46 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0022-3484 
<br/><br/><label>Standard No.: </label>10.1111/j.1600-0765.2005.00842.x  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Anthracenes--pharmacology<br/>Apoptosis--physiology<br/>Butadienes--pharmacology<br/>Calcineurin Inhibitors<br/>Caspases--physiology<br/>Cell Nucleus--ultrastructure<br/>Chromones--pharmacology<br/>Cyclosporine--pharmacology<br/>DNA Fragmentation<br/>Enzyme Inhibitors--pharmacology<br/>Fluorescent Dyes<br/>Imidazoles--pharmacology<br/>Indoles<br/>MAP Kinase Kinase 4--antagonists & inhibitors<br/>Mitogen-Activated Protein Kinase 1--antagonists & inhibitors<br/>Mitogen-Activated Protein Kinase 3--antagonists & inhibitors<br/>Mitogen-Activated Protein Kinase Kinases--antagonists & inhibitors<br/>Morpholines--pharmacology<br/>Myocardium--cytology<br/>Nitriles--pharmacology<br/>Phosphoinositide-3 Kinase Inhibitors<br/>Porphyromonas gingivalis--physiology<br/>Pyridines--pharmacology<br/>Rats<br/>p38 Mitogen-Activated Protein Kinases--antagonists & inhibitors