Yu, Shiguang <br/><br/>
Thyrocytes responding to IFN-gamma are essential for development of lymphocytic spontaneous autoimmune thyroiditis and inhibition of thyrocyte hyperplasia.  [electronic resource] 

 -  Journal of immunology (Baltimore, Md. : 1950)  Jan 2006 
 - 1259-65 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S. 
<br/><br/><label>ISSN: </label>0022-1767 
<br/><br/><label>Standard No.: </label>10.4049/jimmunol.176.2.1259  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adoptive Transfer<br/>Animals<br/>Base Sequence<br/>Chemokines--genetics<br/>Hyperplasia<br/>Interferon-gamma--deficiency<br/>Lymphocytes--immunology<br/>Male<br/>Mice<br/>Mice, Inbred NOD<br/>Mice, Knockout<br/>RNA, Messenger--genetics<br/>Receptors, Interferon--deficiency<br/>Thyroid Gland--immunology<br/>Thyroiditis, Autoimmune--etiology<br/>Interferon gamma Receptor