Harashima, Chie <br/><br/>
Abnormal expression of the G-protein-activated inwardly rectifying potassium channel 2 (GIRK2) in hippocampus, frontal cortex, and substantia nigra of Ts65Dn mouse: a model of Down syndrome.  [electronic resource] 

 -  The Journal of comparative neurology  Feb 2006 
 - 815-33 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0021-9967 
<br/><br/><label>Standard No.: </label>10.1002/cne.20844  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Disease Models, Animal<br/>Down Syndrome--genetics<br/>Frontal Lobe--metabolism<br/>G Protein-Coupled Inwardly-Rectifying Potassium Channels--genetics<br/>Gene Expression Regulation<br/>Hippocampus--metabolism<br/>Immunohistochemistry<br/>In Situ Hybridization<br/>Male<br/>Mice<br/>Mice, Inbred C3H<br/>Mice, Inbred C57BL<br/>Mice, Mutant Strains<br/>Neuronal Plasticity--genetics<br/>Protein Subunits--genetics<br/>RNA, Messenger--analysis<br/>Reverse Transcriptase Polymerase Chain Reaction<br/>Statistics, Nonparametric<br/>Substantia Nigra--metabolism