Hypothyroid phenotype is contributed by mitochondrial complex I inactivation due to translocated neuronal nitric-oxide synthase. [electronic resource]
- The Journal of biological chemistry Feb 2006
- 4779-86 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
0021-9258
10.1074/jbc.M512080200 doi
Animals Cyclin D1--metabolism Cytosol--metabolism Electron Transport Complex I--metabolism Electrons Electrophoresis, Polyacrylamide Gel HSP90 Heat-Shock Proteins--metabolism Hypothyroidism--metabolism Immunoblotting Immunoprecipitation Liver--enzymology MAP Kinase Signaling System Male Microscopy, Immunoelectron Mitochondria--metabolism Mitochondria, Liver--metabolism Models, Chemical NG-Nitroarginine Methyl Ester--pharmacology Nitric Oxide Synthase--metabolism Nitric Oxide Synthase Type I--metabolism Oxidants--metabolism Oxygen--metabolism Peroxynitrous Acid--chemistry Phenotype Protein Isoforms Protein Transport RNA, Messenger--metabolism Rats Rats, Wistar Reactive Oxygen Species--metabolism Reverse Transcriptase Polymerase Chain Reaction Signal Transduction Subcellular Fractions--metabolism Thyroid Hormones--metabolism Transcription, Genetic p38 Mitogen-Activated Protein Kinases--metabolism