Franco, Marķa C

Hypothyroid phenotype is contributed by mitochondrial complex I inactivation due to translocated neuronal nitric-oxide synthase. [electronic resource] - The Journal of biological chemistry Feb 2006 - 4779-86 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

0021-9258

10.1074/jbc.M512080200 doi


Animals
Cyclin D1--metabolism
Cytosol--metabolism
Electron Transport Complex I--metabolism
Electrons
Electrophoresis, Polyacrylamide Gel
HSP90 Heat-Shock Proteins--metabolism
Hypothyroidism--metabolism
Immunoblotting
Immunoprecipitation
Liver--enzymology
MAP Kinase Signaling System
Male
Microscopy, Immunoelectron
Mitochondria--metabolism
Mitochondria, Liver--metabolism
Models, Chemical
NG-Nitroarginine Methyl Ester--pharmacology
Nitric Oxide Synthase--metabolism
Nitric Oxide Synthase Type I--metabolism
Oxidants--metabolism
Oxygen--metabolism
Peroxynitrous Acid--chemistry
Phenotype
Protein Isoforms
Protein Transport
RNA, Messenger--metabolism
Rats
Rats, Wistar
Reactive Oxygen Species--metabolism
Reverse Transcriptase Polymerase Chain Reaction
Signal Transduction
Subcellular Fractions--metabolism
Thyroid Hormones--metabolism
Transcription, Genetic
p38 Mitogen-Activated Protein Kinases--metabolism