Harrington, William R <br/><br/>
Estrogen dendrimer conjugates that preferentially activate extranuclear, nongenomic versus genomic pathways of estrogen action.  [electronic resource] 

 -  Molecular endocrinology (Baltimore, Md.)  Mar 2006 
 - 491-502 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>0888-8809 
<br/><br/><label>Standard No.: </label>10.1210/me.2005-0186  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adaptor Proteins, Signal Transducing--drug effects<br/>Binding, Competitive<br/>Breast Neoplasms--drug therapy<br/>Cell Membrane--metabolism<br/>Cell Proliferation<br/>Cytoplasm--metabolism<br/>Dendrimers<br/>Dose-Response Relationship, Drug<br/>Estradiol--metabolism<br/>Estrogens--metabolism<br/>Extracellular Matrix--metabolism<br/>Extracellular Signal-Regulated MAP Kinases--drug effects<br/>Gene Expression Regulation--drug effects<br/>Genome--drug effects<br/>Humans<br/>Phosphorylation<br/>Polyamines--chemistry<br/>Receptors, Estrogen--metabolism<br/>Rhodamines--chemistry<br/>Shc Signaling Adaptor Proteins<br/>Signal Transduction<br/>Src Homology 2 Domain-Containing, Transforming Protein 1<br/>Tumor Cells, Cultured<br/>src-Family Kinases--drug effects