De Palma, Clara <br/><br/>
Endothelial nitric oxide synthase activation by tumor necrosis factor alpha through neutral sphingomyelinase 2, sphingosine kinase 1, and sphingosine 1 phosphate receptors: a novel pathway relevant to the pathophysiology of endothelium.  [electronic resource] 

 -  Arteriosclerosis, thrombosis, and vascular biology  Jan 2006 
 - 99-105 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1524-4636 
<br/><br/><label>Standard No.: </label>10.1161/01.ATV.0000194074.59584.42  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Calcium--metabolism<br/>Cell Adhesion--drug effects<br/>Cells, Cultured<br/>Endothelium, Vascular--cytology<br/>Enzyme Activation--drug effects<br/>Humans<br/>Nitric Oxide Synthase Type III--metabolism<br/>Phosphatidylinositol 3-Kinases--metabolism<br/>Phosphotransferases (Alcohol Group Acceptor)--metabolism<br/>Proto-Oncogene Proteins c-akt--metabolism<br/>Receptors, Lysosphingolipid--metabolism<br/>Signal Transduction--drug effects<br/>Sphingomyelin Phosphodiesterase--metabolism<br/>Tumor Necrosis Factor-alpha--metabolism<br/>Umbilical Veins--cytology