Gao, Qin <br/><br/>
The mitochondrial permeability transition pore and the Ca2+-activated K+ channel contribute to the cardioprotection conferred by tumor necrosis factor-alpha.  [electronic resource] 

 -  Cytokine  Dec 2005 
 - 199-205 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1043-4666 
<br/><br/><label>Standard No.: </label>10.1016/j.cyto.2005.09.008  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Atractyloside--pharmacology<br/>Coronary Circulation--physiology<br/>Cyclosporine--pharmacology<br/>In Vitro Techniques<br/>Ion Channels--drug effects<br/>L-Lactate Dehydrogenase--biosynthesis<br/>Male<br/>Mitochondria, Heart--drug effects<br/>Mitochondrial Membrane Transport Proteins<br/>Mitochondrial Permeability Transition Pore<br/>Myocardial Contraction--drug effects<br/>Myocardial Infarction--prevention & control<br/>Myocardial Ischemia--drug therapy<br/>Myocardial Reperfusion Injury--prevention & control<br/>Permeability<br/>Potassium Channel Blockers--pharmacology<br/>Potassium Channels, Calcium-Activated--drug effects<br/>Rats<br/>Rats, Sprague-Dawley<br/>Tumor Necrosis Factor-alpha--pharmacology