Kuo, Wei-Wen <br/><br/>
Second-hand smoke-induced cardiac fibrosis is related to the Fas death receptor apoptotic pathway without mitochondria-dependent pathway involvement in rats.  [electronic resource] 

 -  Environmental health perspectives  Oct 2005 
 - 1349-53 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0091-6765 
<br/><br/><label>Standard No.: </label>10.1289/ehp.7479  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis--physiology<br/>Blotting, Western<br/>Caspase 3<br/>Caspases--genetics<br/>Fibrosis--enzymology<br/>Heart Diseases--enzymology<br/>Insulin-Like Growth Factor I--genetics<br/>Male<br/>Mitochondria--physiology<br/>RNA, Messenger--genetics<br/>Rats<br/>Rats, Wistar<br/>Receptor, IGF Type 1--genetics<br/>Reverse Transcriptase Polymerase Chain Reaction<br/>Tobacco Smoke Pollution--adverse effects<br/>fas Receptor--physiology