Kim, Soo Wan <br/><br/>
Increased apical targeting of renal ENaC subunits and decreased expression of 11betaHSD2 in HgCl2-induced nephrotic syndrome in rats.  [electronic resource] 

 -  American journal of physiology. Renal physiology  Mar 2006 
 - F674-87 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Intramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1931-857X 
<br/><br/><label>Standard No.: </label>10.1152/ajprenal.00084.2005  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>11-beta-Hydroxysteroid Dehydrogenase Type 2--genetics<br/>Animals<br/>Disease Models, Animal<br/>Epithelial Sodium Channels<br/>Gene Expression Regulation--drug effects<br/>Gene Expression Regulation, Enzymologic--drug effects<br/>Kidney--drug effects<br/>Kidney Cortex--drug effects<br/>Kidney Medulla--drug effects<br/>Male<br/>Mercuric Chloride--toxicity<br/>Mineralocorticoids--metabolism<br/>Nephrotic Syndrome--chemically induced<br/>Protein Subunits--genetics<br/>Rats<br/>Rats, Inbred BN<br/>Reference Values<br/>Sodium--urine<br/>Sodium Channels--genetics