Hasel, Cornelia <br/><br/>
Parenchymal regression in chronic pancreatitis spares islets reprogrammed for the expression of NFkappaB and IAPs.  [electronic resource] 

 -  Laboratory investigation; a journal of technical methods and pathology  Oct 2005 
 - 1263-75 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0023-6837 
<br/><br/><label>Standard No.: </label>10.1038/labinvest.3700323  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Apoptosis<br/>Apoptosis Regulatory Proteins--biosynthesis<br/>Cell Line, Tumor<br/>Epithelium--metabolism<br/>Fibrosis<br/>Gene Expression Regulation<br/>Humans<br/>I-kappa B Kinase--metabolism<br/>Inhibitor of Apoptosis Proteins--biosynthesis<br/>Insulinoma<br/>Interferon-gamma--physiology<br/>Islets of Langerhans--metabolism<br/>Membrane Glycoproteins--biosynthesis<br/>Microtubule-Associated Proteins--biosynthesis<br/>NF-kappa B--biosynthesis<br/>Neoplasm Proteins--biosynthesis<br/>Pancreatitis, Chronic--metabolism<br/>Protein Subunits--biosynthesis<br/>Receptors, Tumor Necrosis Factor--biosynthesis<br/>Survivin<br/>TNF-Related Apoptosis-Inducing Ligand<br/>Transcription Factor RelA--biosynthesis<br/>Transforming Growth Factor beta--physiology<br/>Tumor Necrosis Factor Decoy Receptors<br/>Tumor Necrosis Factor-alpha--biosynthesis<br/>Up-Regulation