Tanaka, Tetsuhiro <br/><br/>
Cobalt promotes angiogenesis via hypoxia-inducible factor and protects tubulointerstitium in the remnant kidney model.  [electronic resource] 

 -  Laboratory investigation; a journal of technical methods and pathology  Oct 2005 
 - 1292-307 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0023-6837 
<br/><br/><label>Standard No.: </label>10.1038/labinvest.3700328  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Basic Helix-Loop-Helix Transcription Factors--metabolism<br/>Capillaries--drug effects<br/>Capillary Permeability<br/>Cell Movement--drug effects<br/>Cell Proliferation--drug effects<br/>Cells, Cultured<br/>Cobalt--pharmacology<br/>Disease Models, Animal<br/>Endothelial Cells--drug effects<br/>Epithelial Cells--drug effects<br/>Female<br/>Hypoxia--drug therapy<br/>Hypoxia-Inducible Factor 1--metabolism<br/>In Situ Nick-End Labeling<br/>Kidney--blood supply<br/>Kidney Diseases--drug therapy<br/>Kidney Glomerulus--blood supply<br/>Kidney Tubules--blood supply<br/>Neovascularization, Physiologic<br/>Nephrectomy<br/>Rats<br/>Rats, Wistar<br/>Vimentin--metabolism