Salon, C <br/><br/>
E2F1 induces apoptosis and sensitizes human lung adenocarcinoma cells to death-receptor-mediated apoptosis through specific downregulation of c-FLIP(short).  [electronic resource] 

 -  Cell death and differentiation  Feb 2006 
 - 260-72 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1350-9047 
<br/><br/><label>Standard No.: </label>10.1038/sj.cdd.4401739  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adenocarcinoma--chemistry<br/>Apoptosis<br/>Apoptosis Regulatory Proteins--pharmacology<br/>Blotting, Western<br/>CASP8 and FADD-Like Apoptosis Regulating Protein<br/>Caspase 8<br/>Caspases--metabolism<br/>Cell Line, Tumor<br/>Cytotoxicity, Immunologic<br/>DNA--metabolism<br/>Down-Regulation<br/>E2F1 Transcription Factor--genetics<br/>Enzyme Activation<br/>Fas Ligand Protein<br/>Fluorescent Antibody Technique, Indirect<br/>Gene Expression Regulation, Neoplastic<br/>Humans<br/>Intracellular Signaling Peptides and Proteins--genetics<br/>Lung Neoplasms--chemistry<br/>Membrane Glycoproteins--pharmacology<br/>RNA, Messenger--metabolism<br/>Reverse Transcriptase Polymerase Chain Reaction<br/>T-Lymphocytes, Cytotoxic--immunology<br/>TNF-Related Apoptosis-Inducing Ligand<br/>Tumor Necrosis Factor-alpha--pharmacology<br/>Tumor Necrosis Factors--pharmacology