Singh, Kavindra V <br/><br/>
Fsr-independent production of protease(s) may explain the lack of attenuation of an Enterococcus faecalis fsr mutant versus a gelE-sprE mutant in induction of endocarditis.  [electronic resource] 

 -  Infection and immunity  Aug 2005 
 - 4888-94 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0019-9567 
<br/><br/><label>Standard No.: </label>10.1128/IAI.73.8.4888-4894.2005  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Bacterial Proteins--genetics<br/>Endocarditis, Bacterial--enzymology<br/>Enterococcus faecalis--cytology<br/>Gelatinases--genetics<br/>Gene Expression Regulation, Bacterial--physiology<br/>Mutation<br/>Rats<br/>Reverse Transcriptase Polymerase Chain Reaction<br/>Serine Endopeptidases--genetics