Mouse and human resistins impair glucose transport in primary mouse cardiomyocytes, and oligomerization is required for this biological action. [electronic resource]
- The Journal of biological chemistry Sep 2005
- 31679-85 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
0021-9258
10.1074/jbc.M504008200 doi
Animals Biological Transport, Active--physiology Dimerization Exocytosis--physiology Glucose--antagonists & inhibitors Glucose Transporter Type 4 Hormones, Ectopic--genetics Humans Insulin--physiology Male Mice Mice, Inbred C57BL Monosaccharide Transport Proteins--metabolism Muscle Proteins--metabolism Mutation Myocytes, Cardiac--metabolism Phosphorylation Protein Serine-Threonine Kinases--metabolism Protein Transport--physiology Proto-Oncogene Proteins--metabolism Proto-Oncogene Proteins c-akt Resistin Transferrin--metabolism