Dai, R <br/><br/>
Disease-associated fibronectin matrix fragments trigger anoikis of human primary ligament cells: p53 and c-myc are suppressed.  [electronic resource] 

 -  Apoptosis : an international journal on programmed cell death  May 2005 
 - 503-12 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>1360-8185 
<br/><br/><label>Standard No.: </label>10.1007/s10495-005-1880-5  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Anoikis--drug effects<br/>Down-Regulation<br/>Extracellular Matrix--physiology<br/>Fibronectins--physiology<br/>Humans<br/>Inflammation--physiopathology<br/>Peptide Fragments--physiology<br/>Periodontal Ligament--cytology<br/>Proto-Oncogene Proteins c-jun--biosynthesis<br/>Proto-Oncogene Proteins c-myc--biosynthesis<br/>Recombinant Proteins--pharmacology<br/>Tumor Suppressor Protein p53--biosynthesis