Functional genomics identifies a mechanism for estrogen activation of the retinoic acid receptor alpha1 gene in breast cancer cells. [electronic resource]
- Molecular endocrinology (Baltimore, Md.) Jun 2005
- 1584-92 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
0888-8809
10.1210/me.2005-0040 doi
Acetyltransferases--metabolism Base Sequence Breast Neoplasms--metabolism Cell Line, Tumor Chromatin Immunoprecipitation Cloning, Molecular Conserved Sequence Estradiol--metabolism Estrogen Receptor alpha--metabolism Estrogens--metabolism Genome Histone Acetyltransferases Humans Introns Molecular Sequence Data Nuclear Receptor Coactivator 3 Nuclear Receptor Subfamily 1, Group F, Member 1 Oncogene Proteins--metabolism Plasmids--metabolism Promoter Regions, Genetic Protein Binding Receptor Protein-Tyrosine Kinases Receptor Tyrosine Kinase-like Orphan Receptors Receptors, Cell Surface--genetics Receptors, Cytoplasmic and Nuclear Response Elements Reverse Transcriptase Polymerase Chain Reaction Sp1 Transcription Factor--metabolism Trans-Activators--metabolism Transcription, Genetic Transfection