Bulut, Yonca

Mycobacterium tuberculosis heat shock proteins use diverse Toll-like receptor pathways to activate pro-inflammatory signals. [electronic resource] - The Journal of biological chemistry Jun 2005 - 20961-7 p. digital

Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, P.H.S.

0021-9258

10.1074/jbc.M411379200 doi


Animals
Bacterial Proteins--chemistry
Bone Marrow Cells--cytology
Cell Line
Cells, Cultured
Chaperonin 60
Chaperonins--chemistry
DNA, Complementary--metabolism
Dose-Response Relationship, Drug
Endothelium, Vascular--metabolism
Genes, Dominant
HSP70 Heat-Shock Proteins--chemistry
Heat-Shock Proteins--metabolism
Humans
Immune System
Inflammation
Lipopolysaccharide Receptors--chemistry
Macrophages--cytology
Membrane Glycoproteins--metabolism
Mice
Mycobacterium tuberculosis--metabolism
NF-kappa B--metabolism
Peptides--chemistry
Phagocytosis
Receptors, Cell Surface--metabolism
Recombinant Proteins--chemistry
Skin--metabolism
Toll-Like Receptor 2
Toll-Like Receptor 4
Toll-Like Receptors
Transfection