Inhibition of neutrophil apoptosis by TLR agonists in whole blood: involvement of the phosphoinositide 3-kinase/Akt and NF-kappaB signaling pathways, leading to increased levels of Mcl-1, A1, and phosphorylated Bad. [electronic resource]
- Journal of immunology (Baltimore, Md. : 1950) Mar 2005
- 3633-42 p. digital
Publication Type: Journal Article
0022-1767
10.4049/jimmunol.174.6.3633 doi
Apoptosis--drug effects Carrier Proteins--metabolism Caspase 3 Caspases--metabolism DNA-Binding Proteins--metabolism Humans In Vitro Techniques Membrane Glycoproteins--agonists Mitochondria--drug effects Myeloid Cell Leukemia Sequence 1 Protein NF-kappa B--metabolism Neoplasm Proteins--metabolism Neutrophils--cytology Phosphatidylinositol 3-Kinases--metabolism Phosphorylation Protein Serine-Threonine Kinases--metabolism Proto-Oncogene Proteins--metabolism Proto-Oncogene Proteins c-akt Proto-Oncogene Proteins c-bcl-2--metabolism Receptors, Cell Surface--agonists Replication Protein C Signal Transduction Toll-Like Receptor 1 Toll-Like Receptor 2 Toll-Like Receptor 4 Toll-Like Receptor 5 Toll-Like Receptor 7 Toll-Like Receptor 9 Toll-Like Receptors bcl-Associated Death Protein