Mutation of critical GIRK subunit residues disrupts N- and C-termini association and channel function. [electronic resource]
- The Journal of neuroscience : the official journal of the Society for Neuroscience Feb 2005
- 1836-46 p. digital
Publication Type: Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, P.H.S.
1529-2401
10.1523/JNEUROSCI.4783-04.2005 doi
Amino Acid Sequence Amino Acid Substitution Animals COS Cells Carbachol--pharmacology Chlorocebus aethiops Female G Protein-Coupled Inwardly-Rectifying Potassium Channels Gene Expression Hydrophobic and Hydrophilic Interactions Ion Channel Gating--genetics Ion Transport Molecular Sequence Data Mutagenesis, Site-Directed Oocytes Point Mutation Potassium--metabolism Potassium Channels, Inwardly Rectifying--chemistry Protein Interaction Mapping Protein Structure, Tertiary Protein Transport Rats Recombinant Fusion Proteins--chemistry Sequence Deletion Structure-Activity Relationship Transfection Xenopus laevis