Shinmura, Ken <br/><br/>
Prostacyclin attenuates oxidative damage of myocytes by opening mitochondrial ATP-sensitive K+ channels via the EP3 receptor.  [electronic resource] 

 -  American journal of physiology. Heart and circulatory physiology  May 2005 
 - H2093-101 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0363-6135 
<br/><br/><label>Standard No.: </label>10.1152/ajpheart.01003.2004  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adenosine Triphosphate--metabolism<br/>Animals<br/>Cell Survival--drug effects<br/>Epoprostenol--analogs & derivatives<br/>Flavoproteins--metabolism<br/>Fluorescence<br/>Hydrogen Peroxide--pharmacology<br/>Mitochondria--drug effects<br/>Myocytes, Cardiac--drug effects<br/>Oxidants--pharmacology<br/>Oxidative Stress--drug effects<br/>Potassium Channels--metabolism<br/>RNA, Messenger--analysis<br/>Rats<br/>Rats, Sprague-Dawley<br/>Receptors, Prostaglandin E--genetics<br/>Receptors, Prostaglandin E, EP3 Subtype