Qin, Ling <br/><br/>
Parathyroid hormone uses multiple mechanisms to arrest the cell cycle progression of osteoblastic cells from G1 to S phase.  [electronic resource] 

 -  The Journal of biological chemistry  Jan 2005 
 - 3104-11 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0021-9258 
<br/><br/><label>Standard No.: </label>10.1074/jbc.M409846200  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Bone and Bones--metabolism<br/>Cell Cycle<br/>Cell Cycle Proteins--metabolism<br/>Cell Differentiation<br/>Cell Line, Tumor<br/>Cyclic AMP-Dependent Protein Kinases--metabolism<br/>Cyclin D1--metabolism<br/>Cyclin-Dependent Kinase Inhibitor p21<br/>Dual Specificity Phosphatase 1<br/>Female<br/>Femur--pathology<br/>G1 Phase<br/>Immediate-Early Proteins--metabolism<br/>Immunoblotting<br/>Male<br/>Models, Biological<br/>Osteoblasts--metabolism<br/>Parathyroid Hormone--metabolism<br/>Phosphoprotein Phosphatases--metabolism<br/>Phosphorylation<br/>Protein Phosphatase 1<br/>Protein Tyrosine Phosphatases--metabolism<br/>RNA, Messenger--metabolism<br/>Rats<br/>Rats, Sprague-Dawley<br/>S Phase<br/>Time Factors<br/>Up-Regulation