Ohhata, Tatsuya <br/><br/>
X-inactivation is stably maintained in mouse embryos deficient for histone methyl transferase G9a.  [electronic resource] 

 -  Genesis (New York, N.Y. : 2000)  Nov 2004 
 - 151-6 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1526-954X 
<br/><br/><label>Standard No.: </label>10.1002/gene.20077  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Crosses, Genetic<br/>Dosage Compensation, Genetic<br/>Embryo, Mammalian--enzymology<br/>Female<br/>Gene Targeting<br/>Green Fluorescent Proteins--genetics<br/>Histone Methyltransferases<br/>Histone-Lysine N-Methyltransferase--deficiency<br/>Homozygote<br/>Humans<br/>Male<br/>Methylation<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Mice, Transgenic<br/>Protein Methyltransferases<br/>RNA, Long Noncoding<br/>RNA, Untranslated--metabolism<br/>X Chromosome--metabolism