Hitomi, Yuki <br/><br/>
CD72 polymorphisms associated with alternative splicing modify susceptibility to human systemic lupus erythematosus through epistatic interaction with FCGR2B.  [electronic resource] 

 -  Human molecular genetics  Dec 2004 
 - 2907-17 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0964-6906 
<br/><br/><label>Standard No.: </label>10.1093/hmg/ddh318  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adult<br/>Alternative Splicing<br/>Animals<br/>Antigens, CD--genetics<br/>Antigens, Differentiation, B-Lymphocyte--genetics<br/>Base Sequence<br/>COS Cells<br/>Cohort Studies<br/>DNA Primers<br/>Epistasis, Genetic<br/>Female<br/>Genetic Predisposition to Disease<br/>Genotype<br/>Humans<br/>Introns<br/>Male<br/>Middle Aged<br/>Polymorphism, Genetic<br/>Receptors, IgG--genetics<br/>Reverse Transcriptase Polymerase Chain Reaction