Pietrzykowski, Andrzej Z <br/><br/>
Alcohol tolerance in large-conductance, calcium-activated potassium channels of CNS terminals is intrinsic and includes two components: decreased ethanol potentiation and decreased channel density.  [electronic resource] 

 -  The Journal of neuroscience : the official journal of the Society for Neuroscience  Sep 2004 
 - 8322-32 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>1529-2401 
<br/><br/><label>Standard No.: </label>10.1523/JNEUROSCI.1536-04.2004  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Alcoholism<br/>Animals<br/>Calcium--metabolism<br/>Drug Synergism<br/>Drug Tolerance--physiology<br/>Ethanol--pharmacology<br/>Hypothalamo-Hypophyseal System--cytology<br/>Immunohistochemistry<br/>Large-Conductance Calcium-Activated Potassium Channel alpha Subunits<br/>Large-Conductance Calcium-Activated Potassium Channels<br/>Male<br/>Neurons--drug effects<br/>Organ Culture Techniques<br/>Patch-Clamp Techniques<br/>Potassium Channels, Calcium-Activated--drug effects<br/>Presynaptic Terminals--drug effects<br/>Rats<br/>Rats, Sprague-Dawley<br/>Time Factors