Salmond, Robert J <br/><br/>
The B subunit of Escherichia coli heat-labile enterotoxin induces both caspase-dependent and -independent cell death pathways in CD8+ T cells.  [electronic resource] 

 -  Infection and immunity  Oct 2004 
 - 5850-7 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0019-9567 
<br/><br/><label>Standard No.: </label>10.1128/IAI.72.10.5850-5857.2004  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis--drug effects<br/>Bacterial Toxins--chemistry<br/>CD8-Positive T-Lymphocytes--cytology<br/>Caspase Inhibitors<br/>Caspases--metabolism<br/>Cells, Cultured<br/>Enterotoxins--chemistry<br/>Enzyme Inhibitors--pharmacology<br/>Escherichia coli<br/>Escherichia coli Proteins<br/>Female<br/>Guanidines--pharmacology<br/>Membrane Potentials--drug effects<br/>Mice<br/>Mitochondria--drug effects<br/>Nitric Oxide--metabolism<br/>Nitric Oxide Synthase--metabolism<br/>Protein Subunits--chemistry<br/>Protein Transport<br/>Proto-Oncogene Proteins--metabolism<br/>Proto-Oncogene Proteins c-bcl-2--metabolism<br/>Proton-Motive Force--drug effects<br/>bcl-2-Associated X Protein