Rucha, A
Heterologous desensitization of insulin secretion by GIP (glucose-dependent insulinotropic peptide) in INS-1 cells: the significance of Galphai2 and investigations on the mechanism involved. [electronic resource]
- Cell biochemistry and function
- 205-12 p. digital
Publication Type: Journal Article
ISSN: 0263-6484
Standard No.: 10.1002/cbf.1134 doi
Subjects--Topical Terms: 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine--analogs & derivatives 8-Bromo Cyclic Adenosine Monophosphate--analogs & derivatives Adrenergic alpha-Agonists--pharmacology Animals Brimonidine Tartrate Cell Line, Tumor Cyclic AMP-Dependent Protein Kinases--antagonists & inhibitors Enzyme Inhibitors--pharmacology GTP-Binding Protein alpha Subunit, Gi2 GTP-Binding Protein alpha Subunits, Gi-Go--genetics Gastric Inhibitory Polypeptide--pharmacology Gene Expression--drug effects Glucagon--metabolism Glucagon-Like Peptide 1 Immunosuppressive Agents--pharmacology Insulin--metabolism Insulin Secretion Islets of Langerhans--cytology Peptide Fragments--metabolism Protein Precursors--metabolism Proto-Oncogene Proteins--genetics Quinoxalines--pharmacology RNA, Messenger--metabolism Rats Second Messenger Systems--drug effects Tacrolimus--pharmacology Thionucleotides--pharmacology