Imahashi, Kenichi <br/><br/>
Transgenic expression of Bcl-2 modulates energy metabolism, prevents cytosolic acidification during ischemia, and reduces ischemia/reperfusion injury.  [electronic resource] 

 -  Circulation research  Oct 2004 
 - 734-41 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>1524-4571 
<br/><br/><label>Standard No.: </label>10.1161/01.RES.0000143898.67182.4c  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adenosine Triphosphate--metabolism<br/>Anaerobiosis<br/>Animals<br/>Apoptosis<br/>Blotting, Western<br/>Cytosol--chemistry<br/>Energy Metabolism--drug effects<br/>Female<br/>Gene Expression Regulation<br/>Genes, bcl-2<br/>Glycolysis<br/>Humans<br/>Hydrogen-Ion Concentration<br/>Male<br/>Mice<br/>Mice, Transgenic<br/>Mitochondria, Heart--drug effects<br/>Myocardial Contraction<br/>Myocardial Infarction--pathology<br/>Myocardial Ischemia--metabolism<br/>Myocardial Reperfusion Injury--metabolism<br/>Nuclear Magnetic Resonance, Biomolecular<br/>Oligomycins--pharmacology<br/>Phosphocreatine--metabolism<br/>Porins--metabolism<br/>Proto-Oncogene Proteins c-bcl-2--biosynthesis<br/>Proton-Translocating ATPases--physiology<br/>Recombinant Fusion Proteins--physiology<br/>Voltage-Dependent Anion Channels