van den Hoff, Maurice J B <br/><br/>
Increased cardiac workload by closure of the ductus arteriosus leads to hypertrophy and apoptosis rather than to hyperplasia in the late fetal period.  [electronic resource] 

 -  Naunyn-Schmiedeberg's archives of pharmacology  Sep 2004 
 - 193-202 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0028-1298 
<br/><br/><label>Standard No.: </label>10.1007/s00210-004-0955-0  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Anti-Inflammatory Agents, Non-Steroidal--toxicity<br/>Antimetabolites--pharmacology<br/>Apoptosis<br/>Body Weight<br/>Bromodeoxyuridine--pharmacology<br/>Cell Death<br/>Constriction, Pathologic--chemically induced<br/>Ductus Arteriosus--pathology<br/>Fetal Heart--growth & development<br/>Gene Expression Regulation, Developmental<br/>Hyperplasia<br/>Hypertrophy--etiology<br/>In Situ Hybridization<br/>In Situ Nick-End Labeling<br/>Indomethacin--toxicity<br/>Organ Size<br/>Rats<br/>Rats, Wistar