Yeh, Chi-Hsiao <br/><br/>
Nitric oxide attenuates cardiomyocytic apoptosis via diminished mitochondrial complex I up-regulation from cardiac ischemia-reperfusion injury under cardiopulmonary bypass.  [electronic resource] 

 -  The Journal of thoracic and cardiovascular surgery  Aug 2004 
 - 180-8 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0022-5223 
<br/><br/><label>Standard No.: </label>10.1016/j.jtcvs.2003.11.056  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis--drug effects<br/>Cardiopulmonary Bypass--adverse effects<br/>Dogs<br/>Electron Transport Complex I--drug effects<br/>Molsidomine--analogs & derivatives<br/>Myocardial Reperfusion Injury--enzymology<br/>Myocytes, Cardiac<br/>Nitric Oxide--therapeutic use<br/>Nitric Oxide Donors--therapeutic use<br/>Up-Regulation