Okajima, Kenji

Neutrophil elastase contributes to the development of ischemia-reperfusion-induced liver injury by decreasing endothelial production of prostacyclin in rats. [electronic resource] - American journal of physiology. Gastrointestinal and liver physiology Dec 2004 - G1116-23 p. digital

Publication Type: Journal Article

0193-1857

10.1152/ajpgi.00061.2004 doi


6-Ketoprostaglandin F1 alpha--metabolism
Animals
Capillary Permeability--drug effects
Cephalosporins--antagonists & inhibitors
Chemokines, CXC--metabolism
Cyclooxygenase Inhibitors--pharmacology
Endothelium--metabolism
Enzyme Inhibitors--pharmacology
Epoprostenol--biosynthesis
Glycine--analogs & derivatives
Iloprost--pharmacology
Indomethacin--pharmacology
Intercellular Signaling Peptides and Proteins--metabolism
Leukocyte Elastase--antagonists & inhibitors
Liver--drug effects
Liver Circulation--drug effects
Male
NG-Nitroarginine Methyl Ester--pharmacology
Nitric Oxide--metabolism
Nitric Oxide Synthase--antagonists & inhibitors
Nitric Oxide Synthase Type III
Peroxidase--metabolism
Rats
Rats, Wistar
Reperfusion Injury--enzymology
Sulfonamides--antagonists & inhibitors
Transaminases--metabolism
Vasodilator Agents--pharmacology