Okajima, Kenji
Neutrophil elastase contributes to the development of ischemia-reperfusion-induced liver injury by decreasing endothelial production of prostacyclin in rats. [electronic resource]
- American journal of physiology. Gastrointestinal and liver physiology Dec 2004
- G1116-23 p. digital
Publication Type: Journal Article
ISSN: 0193-1857
Standard No.: 10.1152/ajpgi.00061.2004 doi
Subjects--Topical Terms: 6-Ketoprostaglandin F1 alpha--metabolism Animals Capillary Permeability--drug effects Cephalosporins--antagonists & inhibitors Chemokines, CXC--metabolism Cyclooxygenase Inhibitors--pharmacology Endothelium--metabolism Enzyme Inhibitors--pharmacology Epoprostenol--biosynthesis Glycine--analogs & derivatives Iloprost--pharmacology Indomethacin--pharmacology Intercellular Signaling Peptides and Proteins--metabolism Leukocyte Elastase--antagonists & inhibitors Liver--drug effects Liver Circulation--drug effects Male NG-Nitroarginine Methyl Ester--pharmacology Nitric Oxide--metabolism Nitric Oxide Synthase--antagonists & inhibitors Nitric Oxide Synthase Type III Peroxidase--metabolism Rats Rats, Wistar Reperfusion Injury--enzymology Sulfonamides--antagonists & inhibitors Transaminases--metabolism Vasodilator Agents--pharmacology