Greve, Bernhard <br/><br/>
Dissimilar background genes control susceptibility to autoimmune disease in the context of different MHC haplotypes: NOD.H-2(s) congenic mice are relatively resistant to both experimental autoimmune encephalomyelitis and type I diabetes.  [electronic resource] 

 -  European journal of immunology  Jul 2004 
 - 1828-38 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0014-2980 
<br/><br/><label>Standard No.: </label>10.1002/eji.200425116  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Animals, Congenic<br/>CD4-Positive T-Lymphocytes--immunology<br/>Cells, Cultured<br/>Cyclophosphamide--pharmacology<br/>Cytokines--biosynthesis<br/>Diabetes Mellitus, Type 1--chemically induced<br/>Encephalomyelitis, Autoimmune, Experimental--genetics<br/>Genetic Predisposition to Disease--genetics<br/>Haplotypes--genetics<br/>Histocompatibility Antigens--genetics<br/>Immunization<br/>Major Histocompatibility Complex--genetics<br/>Mice<br/>Mice, Inbred NOD<br/>Myelin Proteolipid Protein--chemistry<br/>T-Lymphocytes--immunology