Zhou, Feng-Quan
NGF-induced axon growth is mediated by localized inactivation of GSK-3beta and functions of the microtubule plus end binding protein APC. [electronic resource]
- Neuron Jun 2004
- 897-912 p. digital
Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
ISSN: 0896-6273
Standard No.: 10.1016/j.neuron.2004.05.011 doi
Subjects--Topical Terms: Adenomatous Polyposis Coli--metabolism Analysis of Variance Animals Antineoplastic Agents--pharmacology Axons--drug effects Blotting, Western--methods Cell Count Cells, Cultured Cytoskeletal Proteins--metabolism Dose-Response Relationship, Drug Drug Interactions Embryo, Mammalian Enzyme Inhibitors--pharmacology Fluorescent Antibody Technique--methods Ganglia, Spinal--cytology Gene Expression Regulation--genetics Glycogen Synthase Kinase 3--metabolism Glycogen Synthase Kinase 3 beta Green Fluorescent Proteins Growth Cones--drug effects Luminescent Proteins--metabolism Mice Microtubule-Associated Proteins--metabolism Microtubules--metabolism Models, Neurological Mutagenesis, Site-Directed--physiology Nerve Growth Factors--immunology Neurons--cytology Protein Serine-Threonine Kinases--metabolism Proto-Oncogene Proteins--metabolism Proto-Oncogene Proteins c-akt Signal Transduction--genetics Trans-Activators--metabolism Transfection--methods Tubulin--metabolism beta Catenin