Gaster, Michael <br/><br/>
The primary defect in glycogen synthase activity is not based on increased glycogen synthase kinase-3alpha activity in diabetic myotubes.  [electronic resource] 

 -  Biochemical and biophysical research communications  Jul 2004 
 - 1235-40 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0006-291X 
<br/><br/><label>Standard No.: </label>10.1016/j.bbrc.2004.05.109  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adult<br/>Animals<br/>Cells, Cultured<br/>Diabetes Mellitus, Type 2--enzymology<br/>Glycogen Synthase--metabolism<br/>Glycogen Synthase Kinase 3--metabolism<br/>Glycogen Synthase Kinase 3 beta<br/>Humans<br/>Insulin--metabolism<br/>Male<br/>Middle Aged<br/>Muscle Fibers, Skeletal--cytology<br/>Muscle, Skeletal--cytology<br/>Phosphorylation