Bonafé, M <br/><br/>
The different apoptotic potential of the p53 codon 72 alleles increases with age and modulates in vivo ischaemia-induced cell death.  [electronic resource] 

 -  Cell death and differentiation  Sep 2004 
 - 962-73 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1350-9047 
<br/><br/><label>Standard No.: </label>10.1038/sj.cdd.4401415  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adult<br/>Age Factors<br/>Aged<br/>Aged, 80 and over<br/>Alleles<br/>Apoptosis<br/>Arginine<br/>Blotting, Western<br/>Cell Death<br/>Codon<br/>Creatine Kinase--blood<br/>Creatine Kinase, MB Form<br/>Dose-Response Relationship, Drug<br/>Female<br/>Fibroblasts--metabolism<br/>Flow Cytometry<br/>Genes, p53<br/>Genotype<br/>Homozygote<br/>Humans<br/>Immunoprecipitation<br/>Ischemia<br/>Isoenzymes--blood<br/>Leukocytes--metabolism<br/>Lymphocytes--metabolism<br/>Male<br/>Membrane Potentials<br/>Microscopy, Fluorescence<br/>Middle Aged<br/>Myocardial Ischemia--pathology<br/>Oxidative Stress<br/>Polymorphism, Genetic<br/>Proline<br/>Proto-Oncogene Proteins c-bcl-2<br/>Regression Analysis<br/>Serine--chemistry<br/>Time Factors<br/>Transfection<br/>Troponin I--blood<br/>Tumor Suppressor Protein p53--physiology<br/>bcl-X Protein