van de Geijn, Gert-Jan M <br/><br/>
G-CSF receptor truncations found in SCN/AML relieve SOCS3-controlled inhibition of STAT5 but leave suppression of STAT3 intact.  [electronic resource] 

 -  Blood  Aug 2004 
 - 667-74 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0006-4971 
<br/><br/><label>Standard No.: </label>10.1182/blood-2003-08-2913  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Amino Acid Sequence<br/>Animals<br/>Binding Sites<br/>DNA-Binding Proteins--antagonists & inhibitors<br/>Disease Models, Animal<br/>Humans<br/>Leukemia, Myeloid, Acute--genetics<br/>Mice<br/>Mice, Mutant Strains<br/>Milk Proteins<br/>Neutropenia--genetics<br/>Receptors, Granulocyte Colony-Stimulating Factor--genetics<br/>Repressor Proteins--genetics<br/>STAT3 Transcription Factor<br/>STAT5 Transcription Factor<br/>Sequence Deletion<br/>Signal Transduction<br/>Suppressor of Cytokine Signaling 3 Protein<br/>Suppressor of Cytokine Signaling Proteins<br/>Trans-Activators--antagonists & inhibitors<br/>Transcription Factors--genetics<br/>Tyrosine<br/>src Homology Domains