Hong, Feng <br/><br/>
Transgenic mice overexpressing cyclophilin A are resistant to cyclosporin A-induced nephrotoxicity via peptidyl-prolyl cis-trans isomerase activity.  [electronic resource] 

 -  Biochemical and biophysical research communications  Apr 2004 
 - 1073-80 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0006-291X 
<br/><br/><label>Standard No.: </label>10.1016/j.bbrc.2004.02.160  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Antioxidants--metabolism<br/>Cyclophilin A--genetics<br/>Cyclosporine--pharmacology<br/>Dose-Response Relationship, Drug<br/>Drug Resistance--physiology<br/>Kidney--metabolism<br/>Mice<br/>Mice, Transgenic<br/>Peptidylprolyl Isomerase--metabolism<br/>Recombinant Proteins--metabolism<br/>Survival Rate