Cheema, Zulfiqar F <br/><br/>
The extracellular matrix, p53 and estrogen compete to regulate cell-surface Fas/Apo-1 suicide receptor expression in proliferating embryonic cerebral cortical precursors, and reciprocally, Fas-ligand modifies estrogen control of cell-cycle proteins.  [electronic resource] 

 -  BMC neuroscience  Mar 2004 
 - 11 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>1471-2202 
<br/><br/><label>Standard No.: </label>10.1186/1471-2202-5-11  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Anoikis--physiology<br/>Apoptosis--physiology<br/>Blotting, Western<br/>CASP8 and FADD-Like Apoptosis Regulating Protein<br/>Carrier Proteins--biosynthesis<br/>Cell Cycle--physiology<br/>Cell Cycle Proteins--metabolism<br/>Cell Nucleus--metabolism<br/>Cell-Matrix Junctions--metabolism<br/>Cells, Cultured<br/>Cerebral Cortex--cytology<br/>Cyclin-Dependent Kinase Inhibitor p21<br/>Cyclins--metabolism<br/>Estrogens--pharmacology<br/>Extracellular Matrix--metabolism<br/>Fas Ligand Protein<br/>Flow Cytometry<br/>Intracellular Signaling Peptides and Proteins<br/>Membrane Glycoproteins--physiology<br/>Neurons--classification<br/>Proliferating Cell Nuclear Antigen--metabolism<br/>Rats<br/>Rats, Sprague-Dawley<br/>Stem Cells--cytology<br/>Tumor Suppressor Protein p53--metabolism<br/>fas Receptor--metabolism