Andrabi, Shaida A <br/><br/>
Direct inhibition of the mitochondrial permeability transition pore: a possible mechanism responsible for anti-apoptotic effects of melatonin.  [electronic resource] 

 -  FASEB journal : official publication of the Federation of American Societies for Experimental Biology  May 2004 
 - 869-71 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1530-6860 
<br/><br/><label>Standard No.: </label>10.1096/fj.03-1031fje  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis--drug effects<br/>Calcium Signaling--drug effects<br/>Cell Hypoxia<br/>Corpus Striatum--cytology<br/>Infarction, Middle Cerebral Artery--drug therapy<br/>Inhibitory Concentration 50<br/>Ion Channels--antagonists & inhibitors<br/>Ischemic Attack, Transient--drug therapy<br/>Male<br/>Melatonin--pharmacology<br/>Mice<br/>Mitochondria, Liver--drug effects<br/>Mitochondrial Membrane Transport Proteins<br/>Mitochondrial Permeability Transition Pore<br/>Models, Animal<br/>N-Methylaspartate--toxicity<br/>Neuroprotective Agents--pharmacology<br/>Patch-Clamp Techniques<br/>Rats<br/>Rats, Sprague-Dawley<br/>Rats, Wistar<br/>Receptors, N-Methyl-D-Aspartate--drug effects<br/>Reperfusion Injury--drug therapy